Mechanism only, which is the line this course draws against Clinical Medicine and Surgery: how the disease works belongs here, what is done about it belongs there. A cardiac lecture is the hardest place to hold that line, because every condition in it has an obvious treatment — so the build rejects a management term in ANY option, not just in the correct one. One idea organizes the valve half. Stenosis makes the heart do PRESSURE work and regurgitation makes it do VOLUME work, and which chamber hypertrophies, which dilates, and where the pressure backs up to all follow from that. It is worth reasoning from rather than learning valve by valve. Three numbers are worth holding, and they are easy to swap. Seventy-five percent occlusion before coronary flow is compromised; about fifty percent closure before a stenotic valve affects hemodynamics; and thirty minutes from complete coronary occlusion to irreversible necrosis, with inability to contract arriving within a few minutes of that clock starting. No recording exists for this lecture, so every question comes off the slides and every one cites the slide it came from.
Answers reveal only after you choose. Your score and a breakdown by objective appear at the end, with a review of everything you missed.