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PAJ 5500 · Class of 2028

Clinical Medicine and Surgery I · Exam 3

Ear, nose and throat block — Lectures 15 to 17, everything that has to be recallable cold

How to use this: this is a condensed, night-before-the-exam reference, not a replacement for the full study guide — it assumes you've already learned the material and just need the highest-yield facts at a glance. If a term feels unfamiliar, go back to the full guide for the explanation.

The testsobjectives b to f

TermWhat you need to know
WEBERTuning fork on the MIDLINE. Compares BONE conduction between the two ears. Asks WHICH SIDE IS LOUDER.
RINNETuning fork at the EAR then the MASTOID. Compares AIR against BONE in ONE ear.
CONDUCTIVEWeber lateralizes TO the BAD ear. Rinne: BC ≥ AC. Voice stays SOFT. Hearing BETTER in noise.
SENSORINEURALWeber lateralizes AWAY, to the GOOD ear. Rinne: AC > BC — SAME AS NORMAL. Voice LOUD. Hearing WORSE in noise.
WHY WEBER DECIDESA sensorineural Rinne looks exactly like a normal ear. Only WEBER separates them.
THE QUESTION SHE PROMISEDFullness + reduced hearing AFTER A COLD, AMBER effusion behind an INTACT drum with REDUCED MOBILITY → conductive → Weber TO that ear, BC ≥ AC. She also asks it BACKWARDS: given the fork findings, name the diagnosis — the distractors die on LOSS TYPE.
AUDIOMETRY — ALL BY 20sNormal 0–20 · MILD 20–40 · MODERATE 40–60 · SEVERE 60–80 · PROFOUND >80 dB. The prevalence percentages are explicitly NOT to be memorized.
TYMPANOGRAMA normal · B FLAT = fluid or perforation · C peak NEGATIVE = eustachian tube dysfunction · As SHALLOW/stiff = ossicular FIXATION, tympanosclerosis · Ad DEEP = ossicular DISCONTINUITY, monomeric drum.
VESTIBULARELECTRONYSTAGMOGRAPHY = gold standard, one ear at a time. MRI WITH GADOLINIUM = gold standard for RETROCOCHLEAR. DIX-HALLPIKE diagnoses positional vertigo; EPLEY treats it.

Vertigo, by how long it lastsshe said definitely know this

TermWhat you need to know
SECONDS (10–60)BENIGN PAROXYSMAL POSITIONAL VERTIGO. On rolling over. Hearing NOT affected, NO tinnitus. Beyond a MINUTE → think again. Dix-Hallpike diagnoses, EPLEY treats.
MINUTES TO HOURSMÉNIÈRE’S. Tetrad: vertigo + FLUCTUATING LOW-FREQUENCY sensorineural loss + LOW-TONE tinnitus + FULLNESS. Hearing IS affected. RULE OUT SYPHILIS — indistinguishable.
DAYS TO WEEKS, hearing AFFECTEDLABYRINTHITIS. Inflammation of the membranous labyrinth. Viral. Meclizine or diazepam, steroids, antibiotics ONLY if bacterial features.
DAYS TO WEEKS, hearing NORMALVESTIBULAR NEURONITIS. Inflammation of the VESTIBULAR portion of CN VIII. BENIGN and SELF-LIMITING. No hearing change, NO focal neurology.
THE ONE WORDLabyrinthitis vs vestibular neuronitis = does it affect HEARING. That is the whole difference.
CENTRAL — STOPVertigo + FACIAL PARALYSIS, CROSSED sensory loss, GAZE palsy or HORNER = VERTEBROBASILAR. Vertigo + ATAXIA, HEADACHE or facial numbness = CEREBELLAR INFARCT. Both EMERGENT.

Sort the whole blockgenerated from the chart

TermWhat you need to know
CONDUCTIVEEUSTACHIAN TUBE DYSFUNCTION · ACUTE OTITIS MEDIA · OTITIS MEDIA WITH EFFUSION · CHRONIC OTITIS MEDIA · MASTOIDITIS · CERUMEN IMPACTION · CHOLESTEATOMA · TYMPANIC MEMBRANE PERFORATION · FOREIGN BODY OF THE CANAL · OTITIS EXTERNA · MALIGNANT OTITIS EXTERNA · OTOMYCOSIS · OTOSCLEROSIS · CARCINOMA OF THE EAR CANAL · CONDUCTIVE HEARING LOSS · EXOSTOSIS · GLOMUS TUMOR
SENSORINEURALSENSORINEURAL HEARING LOSS · PRESBYCUSIS · OTOTOXICITY · NOISE-INDUCED HEARING LOSS · PERILYMPHATIC FISTULA · AUTOIMMUNE SENSORINEURAL LOSS · SYPHILITIC SENSORINEURAL LOSS · AIDS-RELATED SENSORINEURAL LOSS · HEREDITARY SENSORINEURAL LOSS · SUDDEN SENSORINEURAL HEARING LOSS · MÉNIÈRE'S DISEASE · LABYRINTHITIS · ACOUSTIC NEUROMA
HEARING NOT AFFECTEDHEMATOMA OF THE EXTERNAL EAR · LACERATIONS AND AVULSION · FOREIGN BODY OF THE AURICLE · KELOID OF THE EAR · BENIGN PAROXYSMAL POSITIONAL VERTIGO · VESTIBULAR NEURONITIS · ISOLATED CEREBELLAR INFARCTION
EMERGENTMALIGNANT OTITIS EXTERNA · CARCINOMA OF THE EAR CANAL · SUDDEN SENSORINEURAL HEARING LOSS · VERTEBROBASILAR INSUFFICIENCY OR OCCLUSION · ISOLATED CEREBELLAR INFARCTION · SINUSITIS WITH URGENT FEATURES · EPISTAXIS — POSTERIOR · NASOPHARYNGEAL CARCINOMA · NECK NEOPLASM — GENERAL · PRIMARY NECK TUMORS — THE LIST · ANAPLASTIC THYROID CARCINOMA · EPIGLOTTITIS (SUPRAGLOTTITIS) · DIPHTHERIA · PERITONSILLAR ABSCESS (QUINSY) · RETROPHARYNGEAL ABSCESS · LUDWIG ANGINA · ERYTHROPLAKIA · ORAL CAVITY AND OROPHARYNGEAL CANCER

The otitis media family

TermWhat you need to know
ACUTE OTITIS MEDIABULGING ERYTHEMATOUS drum, reduced mobility. Otalgia + FEVER + hearing loss. Peak age ~2 YEARS; adults only 3–15%.
ITS CAUSE — the slide is incompleteFAR AND AWAY MOST ARE VIRAL. Bacterial three: S. PNEUMONIAE, H. INFLUENZAE, M. CATARRHALIS. M. catarrhalis has OVERTAKEN H. influenzae because of VACCINATION — ask IMMUNIZATION STATUS.
ITS TREATMENTMOST RESOLVE SPONTANEOUSLY. Bacterial → AMOXICILLIN. RECURRENT = 3 in 6 months or >4 in 12 → TYMPANOSTOMY TUBES.
OTITIS MEDIA WITH EFFUSIONDULL drum, AIR–FLUID LEVEL, reduced mobility. OFTEN ASYMPTOMATIC, found incidentally. Treat on DURATION + degree of hearing loss + effect on SPEECH AND LANGUAGE.
CHRONIC OTITIS MEDIANON-HEALING PERFORATION. Benign = dry. With effusion = serous drainage. SUPPURATIVE = pus. Refer to ENT.
MASTOIDITISSpread into the MASTOID AIR CELLS. Complication of acute otitis media — alongside PERFORATION, LABYRINTHITIS, rarely MENINGITIS.

The canal

TermWhat you need to know
OTITIS EXTERNAPAIN ON MOVING THE TRAGUS. Swimmer. P. AERUGINOSA 38%. Remove debris FIRST, then drops ± steroid. WICK if the canal is closed.
MALIGNANT (NECROTIZING)ELDERLY DIABETIC + PAIN OUT OF PROPORTION + FACIAL NERVE WEAKNESS. >95% P. aeruginosa. IMAGING shows BONY involvement. ANTIPSEUDOMONAL — ciprofloxacin. EMERGENT.
OTOMYCOSISITCH > pain. ASPERGILLUS = “WET NEWSPAPER”. CANDIDA = WHITE CURD. Debris removal + topical antifungal.
CERUMEN IMPACTIONMOST OFTEN SELF-INDUCED BY CLEANING. Irrigate ONLY if the drum is INTACT, body-temperature water. TUBES or PERFORATION → ENT.
FOREIGN BODYDO NOT PUSH DEEPER. Firm → loop/hook. Soft → alligator forceps. ORGANIC SWELLS WHEN WET — no irrigation. INSECT → LIDOCAINE first.
CARCINOMA OF THE CANALOTITIS EXTERNA THAT WILL NOT RESPOND + BLOODY otorrhea + FRIABLE canal. Often MISDIAGNOSED. BIOPSY. EMERGENT.

The drum, pressure and fixation

TermWhat you need to know
EUSTACHIAN TUBE DYSFUNCTIONRETRACTED drum, reduced mobility. Fullness + CRACKLING/POPPING after a cold. Decongestant + intranasal steroid. NO FLYING until it resolves.
BAROTRAUMAFlying or DIVING. HEMOTYMPANUM. Window rupture adds VERTIGO + TINNITUS + SENSORINEURAL loss. MYRINGOTOMY gives instant relief.
TM PERFORATIONTHE PAIN STOPS when it ruptures. CENTRAL vs MARGINAL. Most heal; after acute otitis media as fast as 48–72 HOURS.
CHOLESTEATOMAKERATIN in a RETRACTION POCKET + RECURRENT OTORRHEA WITH NO OTITIS EXTERNA. Not a neoplasm, no cholesterol. CT for extent. SURGERY — it erodes bone.
OTOSCLEROSISSTAPES fixation. Gradual CONDUCTIVE loss with a NORMAL DRUM. HEARS BETTER IN NOISE. Weber TO the affected ear. CT first-line imaging. Stapes prosthesis.
AURICULAR HEMATOMASub-perichondrial. Landmarks LOST. DRAIN WITHIN 7 DAYS — then splint. Re-check at 12–24h because it can appear LATE. Otherwise CAULIFLOWER EAR.

Sensorineural causes worth knowing cold

TermWhat you need to know
PRESBYCUSISCommonest sensorineural loss. BILATERAL, SYMMETRICAL, GRADUAL, HIGH FREQUENCY FIRST. “Hears but cannot make out words.” Screen from 65.
OTOTOXICITYAMINOGLYCOSIDES most ototoxic and most common — monitor peaks. Also FUROSEMIDE, ASPIRIN, PLATINUM. Ototoxic drugs are frequently NEPHROTOXIC too.
NOISE-INDUCEDTEMPORARY threshold shift recovers in 24–48h; repeated exposure makes it PERMANENT. Damage from ~80 dB; PAIN at 120.
SUDDEN SNHLUNILATERAL. A SYNDROME, NOT A DISEASE. Viral or vascular. SAME-DAY ENT REFERRAL — steroids work only in the first weeks.
ACOUSTIC NEUROMAUNILATERAL loss with SPEECH DISCRIMINATION WORSE THAN THE TONES PREDICT. May involve CN V and VII. MRI WITH GADOLINIUM.
PERILYMPHATIC FISTULAAUDIBLE POP then sudden loss + vertigo after STRAINING, DIVING or a BLOW. Round or oval window.
SYPHILISINDISTINGUISHABLE FROM MÉNIÈRE’S and TREATABLE. Order FTA-ABS and MHA-TP; VDRL IS NOT HELPFUL. Antibiotic + systemic steroid.
GLOMUS TUMORPULSATILE TINNITUS + vascular middle ear mass. Can paralyze CN IX, X, XI.
TINNITUS RED FLAGUNILATERAL or PULSATILE is investigated. No drug beats placebo; masking and biofeedback may help.

Nose and sinusesLecture 17

TermWhat you need to know
ACUTE SINUSITISUNDER 4 WEEKS. 90–98% VIRAL; only 0.5–2% superinfect. Pain WORSE BENDING FORWARD, localizes to the sinus. 1 in 8 adults; 5th leading reason antibiotics are prescribed.
BACTERIAL — the 5 featuresDOUBLE WORSENING after 5–6 days · ≥10 DAYS persistent · persistent PURULENT discharge · UNILATERAL tooth/facial pain or maxillary tenderness · FEVER.
PAIN IS THE DISCRIMINATORPain occurs ONLY in bacterial and fungal sinusitis and is REPRODUCIBLE ON PALPATION. A common cold is not.
DISCHARGE COLORYellow/green = LEAST helpful. Clear = viral or allergic. Yellow AND putrid = bacterial. BLACK = FUNGUS. Rust = possible S. pneumoniae.
SINUSITIS DIAGNOSTICSNO test separates viral from bacterial. Routine radiography DISCOURAGED — 3+ clinical findings match imaging accuracy. CT for recurrence, treatment failure, or suspected EXTRASINUS involvement.
SINUSITIS TREATMENTMost improve WITHOUT antibiotics. Symptomatic: decongestants, NSAIDs, lavage, intranasal steroids. Bacterial → AMOXICILLIN/CLAVULANATE. Penicillin allergy → DOXYCYCLINE or moxifloxacin. Influenza → oseltamivir 5 days if >13.
URGENT IN SINUSITISDIPLOPIA or visual disturbance · PERIORBITAL swelling/erythema · ALTERED MENTAL STATUS. Get the CT.
CHRONIC SINUSITISOVER 12 WEEKS. Impaired MUCOCILIARY CLEARANCE → REPEATED infections, not one persistent one. Oral steroids + 2 weeks amoxicillin/clavulanate; courses often 3–4 weeks. ENT for surgery, allergy for skin testing.
CHRONIC FUNGALASPERGILLUS, noninvasive, immunocompetent. Mild disease cured by ENDOSCOPIC SURGERY WITHOUT ANTIFUNGALS. Fungus ball → surgery, antifungals ONLY if bony erosion. Allergic form: polyps + asthma + PEANUT-BUTTER mucus.
DEVIATED SEPTUMOne passage smaller. Congestion → ANOSMIA; severe = OSA, snoring, facial pain, RECURRENT NOSEBLEEDS. Treatment SEPTOPLASTY.
PERFORATED SEPTUMINTRANASAL STEROIDS or COCAINE — chronic ischemia. Rarely GRANULOMATOSIS WITH POLYANGIITIS or secondary SYPHILIS. Treat the cause; else septoplasty.
SEPTAL HEMATOMABetween septum and PERICHONDRIUM. Usually TRAUMA; associated with nasal fracture. DRAIN via intranasal incision under general anesthesia.
EPISTAXIS — ANTERIOR90%, from KIESSELBACH’S PLEXUS. Commonest cause = the patient’s finger. Peaks <10 and 45–65; WINTER.
EPISTAXIS — POSTERIORSPHENOPALATINE ARTERY. Significant hemorrhage. HIGHER RISK because of ASPIRATION and subsequent infection.
STOPPING THE BLEEDBlow out clots → spray OXYMETAZOLINE → PINCH THE ALAE 10 MINUTES CONTINUOUSLY. Sit up, lean FORWARD. Cold compress. Then tampons, packing, balloon catheters, thrombogenic foams. DO NOT BLOW THE NOSE.
EPISTAXIS WORKUPPT/INR is NOT routine — only if anticoagulated. Hematocrit + type and crossmatch if massive, with 2 large-bore IV lines.
NASAL FOREIGN BODYUNILATERAL PURULENT FOUL-SMELLING discharge in a young child. Floor of the passage under the INFERIOR turbinate, or in front of the MIDDLE turbinate. Visualization is the diagnosis.
NASAL FRACTURE — no x-ray ifALL FOUR: tenderness/swelling ISOLATED to the bony bridge · breathes through EACH naris · nose STRAIGHT, no septal deviation · NO SEPTAL HEMATOMA. Otherwise film it. Initial care: ICE + head of bed up.
NASAL POLYPSGRAY, GLISTENING masses → congestion, thick discharge, ANOSMIA. Asthma 20–50%, allergic fungal sinusitis 85%, aspirin intolerance 8–20%, alcohol intolerance 50%, Churg-Strauss 50%, CF 5–44%. EVALUATE EVERY CHILD WITH MULTIPLE POLYPS FOR CF AND ASTHMA — chloride sweat test. Surgery gives only TEMPORARY relief.
ALLERGIC RHINITISCLEAR discharge from BOTH nostrils, BLUISH hue, edematous mucosa, ± polyps. 80% end up on 2+ medicines. Antihistamine + leukotriene inhibitor at night + intranasal steroid; ipratropium; immunotherapy. Allergy creates the environment for infection but is not itself an -itis.
NASOPHARYNGEAL CARCINOMANECK MASS + DIPLOPIA + facial numbness + headache. EBV, HPV, smoking; endemic SOUTHERN CHINA; 2–3× more in males. ENT + endoscopic guided biopsy.

Neck massesLecture 18

TermWhat you need to know
THINK MALIGNANCY WHENNo infectious origin · >2 WEEKS · >1.5 CM · FIRM, non-cystic, NON-TENDER, immobile · AGE >40 · tobacco + alcohol · ULCERATION. Under 40 most are inflammatory — except HODGKIN LYMPHOMA.
FOUR RULES OF THUMBPresent for YEARS = benign (lipoma, cyst) · RAPIDLY growing = infectious if other symptoms, LYMPHOMA if not · FLUCTUANT = cystic · PULSATILE or BRUIT = VASCULAR.
MIDLINE vs LATERALMIDLINE → THYROGLOSSAL DUCT CYST (moves with tongue protrusion), DERMOID CYST, THYROID. LATERAL → BRANCHIAL CLEFT CYST (anterior border of SCM), lymphadenopathy, cystic hygroma.
THE TWO CONGENITAL SIGNSTHYROGLOSSAL DUCT CYST moves VERTICALLY with SWALLOWING or TONGUE PROTRUSION — pathognomonic, and why the SISTRUNK operation takes the CENTER OF THE HYOID. LYMPHANGIOMA TRANSILLUMINATES.
BRANCHIAL CLEFT CYSTInfected after a URI. AVOID I&D — it makes definitive excision harder; needle aspiration if you must. Rule out HPV-associated SCC in an adult — it can present as a cystic neck mass.
HEMANGIOMAGrows through year 1, involutes at 18–24 months, 90% self-resolve. Enlarges with CRYING. First line if you must treat: PROPRANOLOL.
THE EXPOSURE HISTORIESCATS scratch → Bartonella henselae · CAT FECES / undercooked meat → toxoplasmosis · RABBITS, TICKS → tularemia (STREPTOMYCIN) · UNPASTEURIZED MILK → brucellosis (total-body nodes).
MYCOBACTERIA, THE TWOATYPICAL: children, UNILATERAL, BRAWNY reddish-brown skin, surgical excision. TB (SCROFULA): adults > children, DIFFUSE and BILATERAL, RIPE.
THE WORKUPFNA BIOPSY = STANDARD OF CARE, minimum 4 PASSES; separates neoplasm from inflammation and carcinoma from lymphoma. FNA not EXCISIONAL — excision spills tumor. AVOID IODINE CONTRAST if thyroid cancer is suspected.
THYROID MASSImmobile MIDLINE mass that ELEVATES WITH SWALLOWING. Low TSH + nodule → RADIONUCLIDE SCAN FIRST: HOT = no biopsy (low malignant risk); COLD or WARM = FNA.
THYROID CANCER, THE FOURPAPILLARY 75%, best prognosis, young women · FOLLICULAR 16%, spreads by BLOOD to BONE and LUNG, Hürthle variant worse · MEDULLARY 5%, C cells, CALCITONIN, screen family for MEN · ANAPLASTIC 1%, elderly, death 6–36 months, resistant to everything.
THYROID LYMPHOMANon-Hodgkin B cell, on a background of HASHIMOTO. FNA alone cannot separate it from Hashimoto — needs biopsy. Chemo + radiation, NOT surgery.

Mouth, throat and jawLecture 19

TermWhat you need to know
THE SCRAPE TESTWIPES OFF → CANDIDIASIS. Will NOT scrape off + WHITE → LEUKOPLAKIA (premalignant, 5–20% → SCC). Lacy white lines → LICHEN PLANUS (Wickham striae, 1–4% → SCC). Disappears on STRETCHING → LEUKOEDEMA, a normal variant.
ERYTHROPLAKIALike leukoplakia but RED. 90% are already dysplastic or carcinoma. Red beats white for danger, every time.
HAIRY LEUKOPLAKIALATERAL TONGUE, painless, waxes and wanes. EBV. Strongly associated with HIV — a common EARLY finding.
APHTHOUS vs HERPESAPHTHOUS → NON-KERATINIZED, freely moving mucosa (buccal, labial, non-attached gingiva); yellow-gray center, RED HALO. HERPES → keratinized surfaces, with a 24-hour BURNING PRODROME. Recurrent aphthous = SUTTON DISEASE.
SIALOLITHIASISPain and swelling WITH EATING (salivary colic). 80–90% SUBMANDIBULAR — longer duct, alkaline mucin-rich saliva. RADIOPAQUE. CT most sensitive; digital subtraction sialography most accurate.
SUPPURATIVE SIALADENITISPAROTID, dehydrated post-op or elderly patient. S. aureus. PUS FROM THE DUCT. Rehydrate + antistaphylococcal antibiotics. No improvement in 48 h = presume ABSCESS.
VOCAL CORDSNODULES: BILATERAL, symmetric, junction of anterior 1/3 and posterior 2/3, vocal abuse, SPEECH THERAPY first line. POLYPS: UNILATERAL, smokers, excise — may hide an occult SCC. PAPILLOMATOSIS: HPV 6 and 11, warty, AVOID TRACHEOSTOMY.
HOARSENESS CLOCKACUTE LARYNGITIS = commonest cause, persists ~1 week after the cold clears, NO antibiotics. >2 WEEKS = CHRONIC = not a diagnosis → SCOPE IT for cancer and polyps.
EPIGLOTTITISENT EMERGENCY. Children 4 Ds: DROOLING, DYSPHAGIA, DYSPHONIA, DISTRESS + TRIPOD position. HIB. DO NOT examine the mouth or draw blood — it can complete the obstruction. THUMBPRINT SIGN not needed; laryngoscopy is the gold standard. STRIDOR IS LATE.
CENTOR+1 each: NO COUGH, tonsillar EXUDATE, FEVER, tender anterior cervical NODES. Age 3–14 +1, 15–44 0, ≥45 −1. Negative rapid test → ALWAYS culture. Definitive = ASO. Treat: PENICILLIN VK 10 DAYS.
MONONUCLEOSISFever + tonsillar pharyngitis + cervical adenopathy, ages 15–24, EBV 90%. MONOSPOT falsely negative in WEEK 1 (heterophile antibodies take a week to appear) — slide 84 prints “falsely positive”, which is wrong; Prof. Shah confirmed so in class on 9 September. NO CONTACT SPORTS ~1 MONTH — splenic rupture. GIVING PENICILLIN CAUSES A RASH — that rash is often how the diagnosis gets made.
RHEUMATIC FEVER2–3 WEEKS after untreated GABHS, peak ages 5–15. Cross-reactive antibodies attack heart muscle. This is why a sore throat gets an antibiotic at all.
PERITONSILLAR ABSCESSTRIAD: TRISMUS (most reliable) + UVULAR DEVIATION + DYSPHONIA. “HOT POTATO” voice. Commonest deep head and neck infection. Airway first, then needle aspiration / I&D + amoxicillin-clavulanate or clindamycin.
RETROPHARYNGEAL ABSCESSSURGICAL EMERGENCY. Child <5. WIDENED RETROPHARYNGEAL SPACE on lateral neck XR; CT is gold standard. MEDIASTINITIS carries 50% MORTALITY.
LUDWIG ANGINAEMERGENCY. Floor of mouth + submental + sublingual + submandibular. TONGUE PUSHED UP AND BACK. Usually ODONTOGENIC — dental consult is part of the treatment. PCN + metronidazole; bilateral submental drainage if the airway is threatened.
DIPHTHERIATENACIOUS GRAY MEMBRANE over tonsils and pharynx, unimmunized child. ANTITOXIN from the CDC + PCN or erythromycin 14 days. MYOCARDITIS and cranial neuropathy are the killers.
TMJ DISORDERSSECOND commonest musculoskeletal cause of pain and disability. Women of childbearing age. Three categories: MYOFASCIAL PAIN, INTERNAL DERANGEMENT, ARTHRITIS. The ear symptoms are the trap — tinnitus, fullness, dizziness with a normal ear exam.
ORAL CANCERNON-HEALING ULCER. Tobacco + alcohol; 60–80% of OROPHARYNGEAL is HPV. SCC commonest. REFERRED OTALGIA and ill-fitting dentures in advanced disease. Oral cavity → SURGERY ALONE; oropharynx → SURGERY + RADIOTHERAPY.
SALIVARY NEOPLASMThe SMALLER the gland, the likelier MALIGNANT. Parotid 75–80% benign; minor glands only 35% benign. Commonest benign = PLEOMORPHIC ADENOMA; commonest malignant = MUCOEPIDERMOID CARCINOMA. PAIN or FACIAL NERVE involvement = bad prognosis.